Chronic wasting disease threatens state’s deer
A 100% fatal, transmissible, neurogenerative disease has entered the Mississippi white-tailed deer population, and hunters play a big part in controlling this disease.
Chronic wasting disease, or CWD, is a prion disease that is easily transmissible to deer through saliva, feces, urine or a contaminated environment.
The amount of positive material needed to infect deer 100% of the time is the size of one very fine grain of sand. Conditions are ripe for CWD’s spread when an infected deer congregates with other deer in a small area, such as around a salt lick or feeder.
Bronson Strickland, wildlife specialist with the Mississippi State University Extension Service, said CWD’s almost permanent effects make it dangerously different than other diseases.
“Hemorrhagic disease is viral and common among the deer population, but while it can be catastrophic in certain locations and years, that disease cycle has an endpoint,” Strickland said. “This does not happen with CWD.” CWD lingers, both in terms of how slowly the disease kills individual deer and how slowly the disease spreads through a population. “More importantly, there is no annual end point, such as the arrival of cold weather,” Strickland said. “Once the disease becomes entrenched in a population, it is very difficult, if not impossible, to do anything about it.”
William McKinley, deer program coordinator with the Mississippi Department of Wildlife, Fisheries and Parks, said the disease is similar to Creutzfeldt-Jakob disease and Bovine Spongiform Encephalopathy, or mad cow disease.
Unlike mad cow disease, which transmits to humans at a very low rate, CWD has never been known to transmit to humans.
“CWD infects white-tailed deer in Mississippi, and elk, reindeer, moose and red deer in other areas,” McKinley said. “It’s been found in 26 states and three Canadian provinces, as well as in four other countries.”
CWD was first identified in 1967 in a captive mule deer in Fort Collins, Colorado. There is speculation CWD may have mutated from scrapie, a disease similar to CWD that infects sheep. In CWD’s 16- to 20-month incubation period, infected animal shows no signs of disease.
“When the animal becomes clinical, they begin to stumble, are listless, lose weight and forget common things,” McKinley said. “It is comparable to a sped-up version of dementia in humans.”
Physically, the disease causes holes to form in the brain, and when a hole is formed in a region that controls a function critical to life, the animal dies.
“Animals typically die within 6-8 weeks of showing symptoms,” McKinley said. “They spread the disease even when they show no symptoms.”
After appearing in Colorado and spreading in the West, it was first seen east of the Mississippi River in Wisconsin in 2002. That year, the Department of Wildlife, Fisheries and Parks, which regulates wildlife and hunting in Mississippi, began looking for the disease.
The first positive in the state was detected in February 2018 in Issaquena County. Since then, there have been 83 positive deer reported in the state in eight counties: Issaquena (2), Pontotoc (1), Marshall (20), Benton (56), Panola (1), Tallahatchie (1), Tippah (1) and Alcorn (1).
Strickland said every hunter in Mississippi and surrounding states has two good reasons for sampling the deer they harvest.
“Our state wildlife agencies can’t manage the disease to keep it from spreading if they don’t know where it is, and if you eat your venison, testing it first gives you peace of mind,” Strickland said.
He said the Centers for Disease Control and Prevention report that there have been no reported cases of CWD infection in people, but some animal studies raise concerns that there may be a risk to humans.
To keep CWD numbers in check, wildlife specialists have determined that the best management practices are to lower deer density and eliminate practices that encourage deer to congregate.
“This is why the state’s regulatory agency allows more deer to be harvested in CWD zones and why supplemental feeding is eliminated in these areas,” Strickland said. “High concentrations of infectious prions are found in saliva. “Supplemental feeding does not cause CWD, but it facilitates its spread as deer concentrate in certain areas, such as around a feeder,” he said.
Infection rates in other states have shown that the disease does not go away and spreads widely if left unmanaged. Without a cure, CWD is devastating to the deer population and to hunters who depend on deer for food or sport.
“If ignored, the disease will reach the point of no return in localized areas,” Strickland said.
McKinley said inactivity is not an option. “We must change our hunting and deer management behavior, because if we do nothing, we end up with prevalence so high that the disease manages the herd for us,” he said. “Our goal is to keep CWD at a low prevalence in Mississippi.”